Acute Care Hospital Products
NASDAQ: MDCO
For patients presenting to the emergency department (ED) who require rapid blood pressure (BP) control, the goal of therapy is a prompt, smooth reduction in BP while avoiding excessive drops.1 Cleviprex®, with its rapid onset and offset of effect, ultrashort half-life of ~1 minute, metabolism independent of the liver and kidney, and reliable transition to oral antihypertensive therapy, provides a rapid, predictable option for BP control in patients in the ED.2
In a clinical study of 126 patients presenting to the ED or intensive care unit with severe hypertension (VELOCITY trial), Cleviprex rapidly lowered BP to an individualized target BP in ~90% of patients, and the median time to achieve target BP was 10.9 minutes.3
Of patients eligible for transition to oral therapy, 98% (115/118) did so successfully within 6 hours of discontinuing Cleviprex. Overshoot was infrequent (1.6%; 2 of 126) and BP was monitored successfully with a BP cuff and did not require an arterial line in the majority (125 of 126) of patients.
In clinical trials of 215 patients with preoperative or postoperative hypertension (ESCAPE trials), Cleviprex decreased SBP by at least 15% within
30 minutes in more than 90% of patients.4,5 Cleviprex also demonstrated comparable safety and efficacy to 3 other IV antihypertensive treatments (nitroglycerin, sodium nitroprusside, and nicardipine) in clinical trials comprised of 1,512 cardiac surgery patients who required perioperative or postoperative BP reduction (ECLIPSE trials).6
Cleviprex is intended for intravenous use. Titrate drug depending on the response of the individual patient to achieve the desired blood pressure reduction. Monitor blood pressure and heart rate continually during infusion, and then until vital signs are stable. Patients who receive prolonged Cleviprex infusions and are not transitioned to other antihypertensive therapies should be monitored for the possibility of rebound hypertension for at least 8 hours after the infusion is stopped.
Cleviprex is contraindicated in patients with allergies to soybeans, soy products, eggs, or egg products; defective lipid metabolism such as pathologic hyperlipemia, lipoid nephrosis, or acute pancreatitis if it is accompanied by hyperlipidemia; and in patients with severe aortic stenosis.
Hypotension and reflex tachycardia are potential consequences of rapid upward titration of Cleviprex. Dihydropyridine calcium channel blockers can produce negative inotropic effects and exacerbate heart failure. Monitor heart failure patients carefully. Cleviprex gives no protection against the effects of abrupt beta-blocker withdrawal.
Most common adverse reactions (> 2%) are headache, nausea, and vomiting.
Cleviprex should be used during pregnancy only if the potential benefit justifies the potential risk to the fetus.
Maintain aseptic technique while handling Cleviprex. Cleviprex contains phospholipids and can support microbial growth. Do not use if contamination is suspected. Once the stopper is punctured, use and discard within 4 hours.
Please see full Prescribing Information.
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